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Types of color blindness

Switch the scene below between seven forms of color vision deficiency to see what changes. Each card lists the cone involved, how common it is, and which colors get confused.

See it as
A traffic light, ripe and unripe tomatoes with fruit, and a bar chart in a common default palette. Typical color vision.

The seven types at a glance

  • Protanopia

    Red-green

    L (red) cones missing

    Men≈1.0%
    Women≈0.02%

    Reds look darker and can read as brown or black.

    Pairs that move closest (ΔE before → after)
    • Orange / Olive: 33 → 4
    • Green / Orange: 51 → 7
    • Pink / Teal: 55 → 11
  • Protanomaly

    Red-green

    L (red) cones shifted

    Men≈1.1%
    Women≈0.03%

    A milder version of protanopia; severity varies a lot.

    Pairs that move closest (ΔE before → after)
    • Orange / Olive: 33 → 11
    • Green / Orange: 51 → 17
    • Orange / Lime: 41 → 15
  • Deuteranopia

    Red-green

    M (green) cones missing

    Men≈1.3%
    Women≈0.01%

    Reds, oranges, yellows and greens fall onto one yellow-brown axis.

    Pairs that move closest (ΔE before → after)
    • Orange / Lime: 41 → 4
    • Teal / Magenta: 55 → 7
    • Red / Green: 69 → 9
  • Deuteranomaly

    Red-green

    M (green) cones shifted

    Men≈4.6%
    Women≈0.4%

    The most common type. Many people only find out from a screening test.

    Pairs that move closest (ΔE before → after)
    • Orange / Lime: 41 → 14
    • Red / Green: 69 → 26
    • Red / Olive: 50 → 21
  • Tritanopia

    Blue-yellow

    S (blue) cones missing

    Menvery rare
    Womenvery rare

    Blue and green, and yellow and pink, become hard to separate.

    Pairs that move closest (ΔE before → after)
    • Pink / Yellow: 48 → 13
    • Orange / Pink: 34 → 9
    • Green / Blue: 54 → 18
  • Tritanomaly

    Blue-yellow

    S (blue) cones weak

    Menvery rare
    Womenvery rare

    Inherited on chromosome 7, so it affects men and women equally.

    Pairs that move closest (ΔE before → after)
    • Orange / Pink: 34 → 21
    • Orange / Magenta: 47 → 31
    • Sky blue / Lime: 44 → 30
  • Achromatopsia

    No color

    No working cones

    Men≈1 in 30,000
    Women≈1 in 30,000

    Only brightness is seen. Usually comes with light sensitivity and low sharpness.

    Pairs that move closest (ΔE before → after)
    • Sky blue / Lime: 44 → 0
    • Red / Magenta: 26 → 0
    • Green / Gray: 26 → 1

Simulations use the Machado, Oliveira and Fernandes (2009) model on an sRGB screen. They approximate, and real experiences differ from person to person. Nothing on this page can diagnose a color vision deficiency; for that, see an optometrist or ophthalmologist.

How color vision works, briefly

Most people have three kinds of cone cell in the retina. L cones respond most to long wavelengths (reds), M cones to medium (greens) and S cones to short (blues). The brain compares their signals to work out color. Color blindness, more precisely called color vision deficiency, happens when one cone type is missing or responds at the wrong wavelengths.

That gives three families, named after the Greek for first, second and third:

FamilyCone affectedMissing coneShifted cone
ProtanL (red)ProtanopiaProtanomaly
DeutanM (green)DeuteranopiaDeuteranomaly
TritanS (blue)TritanopiaTritanomaly

"-opia" means the cone type is absent, so the person sees with two cone types (dichromacy). "-anomaly" means all three are present but one is shifted, so colors are weaker rather than gone. A seventh condition, achromatopsia, means no cones work at all and the world is seen in shades of gray.

Red-green: by far the most common

Protan and deutan types together are what people usually mean by color blindness. The genes for the L and M cones sit on the X chromosome. Men have one X, so a single altered copy is enough; women need two. That is why commonly cited estimates for people of Northern European ancestry are around 8% of men but only about 0.5% of women. Rates are lower in many other populations.

Deuteranomaly alone accounts for more than half of all cases. It ranges from barely noticeable to almost as strong as deuteranopia, and many people with a mild form only find out when they fail a screening test.

What changes in practice:

  • Protan types see reds as darker. A red traffic light can look almost black, and red text on a dark background may disappear. In the scene above, protanopia turns the red light into a dim olive (#62571A).
  • Deutan types don't darken reds as much, but red, orange, yellow and green collapse toward one yellow-brown axis. In our chart, the default red and green bars are about ΔE 70 apart for typical vision and under 5 apart with deuteranopia: practically the same color.

Both groups mix up pairs that seem obvious to others, such as orange and lime, or red and olive. The cards above list the three pairs from a test set that move closest together for each type, with the color difference before and after.

Blue-yellow: rare and different

Tritan types involve the S cones, whose gene is on chromosome 7, not the X, so men and women are affected equally. Inherited tritan deficiency is rare; estimates vary widely, from about 1 in 10,000 to far fewer. It can also be acquired: aging of the lens, some eye diseases and certain medications can reduce blue sensitivity. With tritanopia, blue and green become hard to separate, and so do pink and yellow.

Achromatopsia

Complete achromatopsia affects around 1 in 30,000 people. Because the cones handle sharp daytime vision, people with achromatopsia usually also have reduced visual acuity, strong sensitivity to bright light and involuntary eye movements. The simulation shows only the color part: every color becomes its brightness. Sky blue and lime green, for example, end up almost exactly the same gray.

What the simulation can and can't show

The scene uses the model published by Machado, Oliveira and Fernandes in 2009, which is widely used for screen simulations. The anomalous types are shown at a moderate severity. Real experience varies by person, severity, screen and lighting, and no one with a deficiency sees the world as a "broken" version of typical vision; their brain has adapted to it all their life. Use the simulations to understand which colors are risky, not to know exactly what someone sees.

Testing and diagnosis

The color blind test on this site uses Ishihara-style dot plates to screen for red-green and blue-yellow differences, and the hue test checks how finely you can order similar colors. Both are screening tools only. A diagnosis needs an eye care professional, who may use printed plates under standard lighting, arrangement tests or an anomaloscope. There is currently no cure for inherited color vision deficiency. Tinted "color blind glasses" can increase contrast between some colors for some people, but they don't restore normal color vision.

Designing for every type

About one in twelve men will have trouble with red-versus-green signals, so:

  • never use color as the only signal; add labels, icons or patterns,
  • check charts and maps with the color blindness simulator, which runs your own image or palette through the same model,
  • make text meet contrast targets with the contrast checker, since contrast also helps people with low color discrimination.